Skip Navigation

Person Details: David G. Kaufman

Superfund Research Program

David G. Kaufman

University of North Carolina-Chapel Hill
Department of Biochemistry and Biophyscis
514 Brinkhous-Bullitt
CB#7295
Chapel Hill, North Carolina 27559
Phone: 919-966-1396
Email: david.kaufman@pathology.unc.edu

Projects

Publications

2011

2010

  • Asagoshi K, Tano K, Chastain PD, Adachi N, Sonoda E, Kikuchi K, Koyama H, Nagata K, Kaufman DG, Takeda S, Wilson SH, Watanabe M, Swenberg JA, Nakamura J. 2010. FEN1 functions in long patch base excision repair under conditions of oxidative stress in vertebrate cells. Mol Cancer Res 8(2):204-215. doi:10.1158/1541-7786.MCR-09-0253 PMID:20145043 PMCID:PMC2824787
  • Chastain PD, Nakamura J, Rao S, Chu H, Ibrahim J, Swenberg JA, Kaufman DG. 2010. Abasic sites preferentially form at regions undergoing DNA replication. FASEB J 24(10):3674-80. doi:10.1096/fj.09-145276 PMID:20511393 PMCID:PMC2996904

2006

  • Chastain PD, Nakamura J, Swenberg JA, Kaufman DG. 2006. Nonrandom AP site distribution in highly proliferative cells. FASEB J 20(14):2612-4. PMID:17068113

1995

  • Chiao C, Zhang Y, Kaufman DG, Kaufmann WK. 1995. Derivation of phenobarbital-responsive immortal rat hepatocytes. Am J Pathol 146(5):1248-1259. PMID:7747817
  • Chiao C, Zhang Y, Kaufman DG, Kaufmann WK. 1995. Phenobarbital modulates the type of cell death by initiated hepatocytes during deprivation of serum in vitro. Hepatology 22(1):297-303. PMID:7601424
  • Zhang Y, Chiao C, Byrd LL, Kaufman DG, Kaufmann WK. 1995. Liver tumor promotion and the suppression of p53-dependent cell cycle checkpoint function. In: Liver Regeneration and Carcinogenesis: molecular and cellular mechanisms. Academic Press,
Back
to Top
Last Reviewed: October 07, 2024